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@Waira @JohnEMad salicylic acid does initiate a SAR response but this does not directly help tric production and actually has a negative effect. Here is a reference article study in the arabidopsis test plant but the biosynthesis pathways are conserved across species so the results are relevant.
“The jar1-1 mutant exhibited normal trichome induction following treatment with jasmonic acid, suggesting that adenylation of jasmonic acid is not necessary. Salicylic acid had a negative effect on trichome production and consistently reduced the effect of jasmonic acid, suggesting negative cross-talk between the jasmonate and salicylate-dependent defense pathways. Interestingly, the effect of salicylic acid persisted in the nim1-1mutant, suggesting that the Npr1/Nim1 gene is not downstream of salicylic acid in the negative regulation of trichome production.”
Note that the jar1-1 mutant is lacking the signal pathway for JA which allows the study of this pathway in plants that would otherwise not respond normally to JA.
The most effective method of impacting the trichome/resin production pathways in trichome producing species of plants is through the jasmonic acid pathway. We have tackled this problem with our Precursor 1 nutrient. We don’t supply jasmonic acid (JA) directly, what we have done is to formulate Precursor 1 with components that have been shown in our experiments and in the scientific literature to support the plants natural biosynthesis mechanisms for JA and thereby allowing the plant to regulate its own metabolism but with the added support of making sure the specific resources are available to promote this mechanism. Like a gentle nudge saying we got your feed on point so go forth and make it sticky...
This is something we put a lot of time into and we definitely believe that Precursor 1 is better suited to increase tric development compared to many other products making that claim.
“The jar1-1 mutant exhibited normal trichome induction following treatment with jasmonic acid, suggesting that adenylation of jasmonic acid is not necessary. Salicylic acid had a negative effect on trichome production and consistently reduced the effect of jasmonic acid, suggesting negative cross-talk between the jasmonate and salicylate-dependent defense pathways. Interestingly, the effect of salicylic acid persisted in the nim1-1mutant, suggesting that the Npr1/Nim1 gene is not downstream of salicylic acid in the negative regulation of trichome production.”
Interactive Effects of Jasmonic Acid, Salicylic Acid, and Gibberellin on Induction of Trichomes in Arabidopsis - PMC
Leaf trichomes protect plants from attack by insect herbivores and are often induced following damage. Hormonal regulation of this plant induction response has not been previously studied. In a series of experiments, we addressed the effects of ...
www.ncbi.nlm.nih.gov
Note that the jar1-1 mutant is lacking the signal pathway for JA which allows the study of this pathway in plants that would otherwise not respond normally to JA.
The most effective method of impacting the trichome/resin production pathways in trichome producing species of plants is through the jasmonic acid pathway. We have tackled this problem with our Precursor 1 nutrient. We don’t supply jasmonic acid (JA) directly, what we have done is to formulate Precursor 1 with components that have been shown in our experiments and in the scientific literature to support the plants natural biosynthesis mechanisms for JA and thereby allowing the plant to regulate its own metabolism but with the added support of making sure the specific resources are available to promote this mechanism. Like a gentle nudge saying we got your feed on point so go forth and make it sticky...
This is something we put a lot of time into and we definitely believe that Precursor 1 is better suited to increase tric development compared to many other products making that claim.
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